Neuromodulation et interface cerveau-machine / Société de neurophysiologie


Le 17/10/2019 de 14:30 à 16:00


Président(s): Jean-pascal LEFAUCHEUR
Modérateur(s): Djamel BENSMAIL, Pierre BURBAUD, Bertrand GLIZE, Perrine SEGUIN

Retour Session

C322

L’altération de l’activité oscillatoire corticale participe à la détérioration des patterns du mouvement du membre supérieur chez le patient post-AVC.

Alexandre Chalard (Toulouse, France), David Amarantini (Toulouse, France), Philippe Marque (Toulouse, France), David Gasq (Toulouse, France), Joseph Tisseyre (Toulouse, France)

Objective : Spastic co-contraction is a motor disabling form of muscle over-activity occurring after stroke, contributing to the limitation of the active movement and motor impairment. To date, the cortical mechanisms underlying spastic co-contraction remain to be more fully elucidated. The cortical activity involved in a motor task is usually investigate by movement-related beta desynchronization, which has been linked to the cortical excitability[1]. Indeed, abnormalities in cortical oscillatory activity could be a key mechanism underlying motor impairment after stroke [2]. This study aimed to investigate the association between cortical oscillations and the alterations of the kinematic and muscle activation patterns after stroke.
Fifteen post-stroke patients and nine healthy controls were included. They performed active elbow extensions during which we assessed elbow kinematics, elbow flexors and extensor electromyographic-based muscles activation, and cortical oscillatory activity by computing the movement-related beta desynchronization from electroencephalography.
In stroke patients, movement-related beta desynchronization was decreased by 28.6 ± 8.3 % during elbow extension movement (t22 = 4.66, P